Research
Eotaxin and FGF enhance signaling through an Extracellular signal-related kinase (ERK)-dependent pathway in the pathogenesis of Eosinophilic Esophagitis
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* Corresponding author: Kari C Nadeau knadeau@stanford.edu
1 Stanford School of Medicine, Stanford, CA 94305, USA
2 California Pacific Medical Center, San Francisco, CA 94118, USA
Allergy, Asthma & Clinical Immunology 2010, 6:25 doi:10.1186/1710-1492-6-25
Published: 5 September 2010Additional files
Additional file 1:
Supplementary tables. Table S1a: Demographics of EoE patients. D (distal) and P (proximal) reflect location of the eosinophil count. Eosinophil counts are given as per high powered field (> greater than; = equal to). If negative for any allergies, total IgE less than 10 kU/mL. Table S1b: Demographics of GERD patients Table S1c: Demographics of healthy controls. Table S1 d. Demographics of Crohn's disease and ulcerative colitis patients. Table S2: Treatment emographics of EoE subjects.
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